Researchers find inflammatory response in fatty liver disease is reduced by two tryptophan metabolites from intestine bacteriaMEDFORD/SOMERVILLE,Mass. (May 3, 2018)—Researchers at Tufts University maintain elucidated a mechanism by which the “good” bacteria that reside in our gastrointestinal tract can help protect us from inflammation, or how their disruption (dysbiosis) can increase the susceptibility of the liver to more harmful forms of disease. Their study,now available in the journal Cell Reports, identified two key metabolites produced by the bacteria in mice that modulate inflammation in the host and could ultimately reduce the severity of non-alcoholic fatty liver disease.
Source: tufts.edu